Axial spondyloarthritis

Active Ingredient: Ixekizumab

Indication for Ixekizumab

Population group: only adults (18 years old or older)
Therapeutic intent: Curative procedure

Ankylosing spondylitis (radiographic axial spondyloarthritis)

Taltz is indicated for the treatment of adult patients with active ankylosing spondylitis who have responded inadequately to conventional therapy.

Non-radiographic axial spondyloarthritis

Taltz is indicated for the treatment of adult patients with active non-radiographic axial spondyloarthritis with objective signs of inflammation as indicated by elevated C-reactive protein (CRP) and/or magnetic resonance imaging (MRI) who have responded inadequately to nonsteroidal anti-inflammatory drugs (NSAIDs).

For this indication, competent medicine agencies globally authorize below treatments:

160 mg at Week 0 and thereafter 80 mg every 4 weeks

For:

Dosage regimens

Subcutaneous, 160 milligrams ixekizumab, 1 one dose, over the duration of 4 weeks. Afterwards, subcutaneous, 80 milligrams ixekizumab, once every 4 weeks.

Detailed description

The recommended dose is 160 mg by subcutaneous injection at week 0, followed by 80 mg every 4 weeks.

Consideration should be given to discontinuing treatment in patients who have shown no response after 16 to 20 weeks of treatment. Some patients with initially partial response may subsequently improve with continued treatment beyond 20 weeks.

No dose adjustment is required in subjects aged ≥65 years. There is limited information in subjects aged ≥75 years.

Dosage considerations

If possible, areas of the skin that show psoriasis should be avoided as injection sites.

Active ingredient

Ixekizumab

Ixekizumab is an IgG4 monoclonal antibody that binds with high affinity (<3 pM) and specificity to interleukin 17A (both IL-17A and IL-17A/F). Elevated concentrations of IL-17A have been implicated in the pathogenesis of psoriasis by promoting keratinocyte proliferation and activation, as well as in the pathogenesis of psoriatic arthritis and axial spondyloarthritis by driving inflammation leading to erosive bone damage and pathological new bone formation. Neutralisation of IL-17A by ixekizumab inhibits these actions.

Read more about Ixekizumab

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